Faculty

Faculty in the Female Pelvic Medicine & Reconstructive Surgery Fellowship at University Hospitals include:

Available mentors from the FPMRS faculty
Faculty Name Research and Clinical Interests
Arnold Caplan, PhD Experimentation in musculoskeletal and skin development; development, maturation and aging of cartilage, bone, skin and other mesenchymal tissues; mesenchymal stem cell research
Sherif A. El-Nashar, MD
  • Female pelvic medicine and reconstructive surgery: clinical research in the field of pelvic floor dysfunction, understanding advanced pelvic organ prolapse and, surgical treatment for vaginal fistula and complications related to vaginal placement of mesh for pelvic organ prolapse surgery.
  • Abnormal uterine bleeding: translational and clinical research related to the uterine bleeding etiology, pathogenesis and management. The scope of my research interest includes understanding of menstrual regulation, heavy menstrual bleeding, fibroids, endometriosis, adenomyosis and endometrial cancer.
  • Minimally invasive surgery in gynecology
  • Heath-related quality of life evaluation in women
  • Evidence-based medicine and systematic review and meta-analysis methodology
  • Quality measurements in obstetrics and gynecology
Christopher Flask, PhD Magnetic resonance imaging (MRI) physics; optimization of MRI acquisitions; analyses for the Diffusion Weighted MRI; high-resolution pelvic MRI images for small animal models
Ken Gustafson, PhD Neural engineering and neural prostheses; systems-level neurophysiology and neural control of lower urinary tract and pelvic functions; design and development of neural prostheses that interface with native spinal neural circuitry and restore physiologic function
Joseph Henderson, MD Epidemiology, pathophysiology, and treatment of overactive bladder, neuromodulation for overactive bladder and fecal incontinence, clinical research in the field of pelvic floor dysfunction, understanding and treatment for advanced pelvic organ prolapse and vaginal fistulas, chronic vulvar and vaginal pain syndromes, and advanced minimally invasive surgery
Adonis Hijaz, MD  Mechanisms mediating the balance between injury and recovery following birth trauma, the role of human mesenchymal stem cells in recovery, and evaluation of the effects of aging on the balance and response to stem cell therapy; animal models of FSUI
Michael Lederman, MD Mechanisms of infectious diseases, specifically HIV/AIDS; mechanisms whereby HIV infection induces immune deficiency, strategies to correct and prevent these mechanisms; mucosal defenses in HIV infection; urinary tract infection in diabetes; neurogenic bladder
Sangeeta Mahajan, MD Epidemiology and pathophysiology of overactive bladder; novel applications of botulinum A toxin in the lower urinary tract; impact of overactive bladder symptoms on the quality of life of patients with multiple sclerosis; relationship between painful bladder syndrome and myofascial pelvic pain
Jeffrey Mangel, MD Urinary incontinence; overactive bladder; pelvic organ prolapse; bowel dysfunction; fecal incontinence; vesicovaginal and rectovaginal fistulas; chronic vulvar and vaginal pain syndromes; sacral neuromodulation for overactive bladder syndrome
Sam Mesiano, PhD Molecular biology of estrogen and progesterone action
Robert Miller, PhD CNS neural development; biology of neural diseases including multiple sclerosis, brain tumors and cerebral palsy; CNS lesions on LUT function; cellular and molecular mechanisms that regulate glial cell determination in the developing vertebrate CNS
Robert Pollard, MD Minimally invasive gynecologic surgery techniques
David Sheyn, MD Pathophysiology and treatment of overactive bladder, neuromodulation for overactive bladder and fecal incontinence, application of artificial intelligence to diagnosis and treatment of pelvic floor disorders, epidemiology of pelvic floor disorders, optimization of robotics for urogynecologic surgery
Jerry Silver, PhD Mechanisms involved in neurogenic bladder and spinal cord injury; strategies to overcome inhibitory molecules and axonal dieback after injury in order to promote functional regeneration
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